Continuing Professional Development (CPD) approved by the CPD Certification Service (CPD UK), the British Association for Nutrition and Lifestyle Medicine (BANT) and the Nutritional Therapists of Ireland (NTOI).

Webinar description:

Menopause is often reduced to a hormonal deficiency narrative. Yet, clinicians increasingly recognize that midlife female physiology is a multidimensional transition involving metabolism, detoxification, circadian rhythms, inflammation, mitochondrial integrity, and neuroendocrine signaling. This presentation reframes menopause through a root-cause, systems-biology lens and positions nutrition and lifestyle as foundational drivers of regulation across the endocrine, metabolic, and neuroimmune networks. 

Rather than focusing solely on isolated hormone levels, this talk introduces a sequenced approach to clinical assessment and intervention. Hormone therapy, when appropriate, becomes one tool within a broader, ordered framework that prioritizes restoring physiological coherence before layering on additional inputs. Using a structured progression—from stress and sleep regulation to thyroid-adrenal-mitochondrial support, metabolic flexibility, gut-liver detoxification capacity, and ultimately estrogen and progesterone metabolism—the presentation offers practitioners a clear roadmap for timing interventions in a way that aligns with known mechanisms of hormone action and biotransformation.

Nutrition is integrated throughout as both a molecular and energetic signal, shaping steroidogenesis, receptor sensitivity, mitochondrial function, immune tone, and circadian alignment. Emphasis is placed on nutrient-dense dietary patterns, phytonutrient diversity, melatonin-responsive systems, amino acid and fatty acid substrates, and the emerging role of the “photometabolome” in midlife physiology.

Lifestyle factors—including sleep-wake organization, light exposure, stress modulation, movement, environmental toxicant load, and psychosocial context—are examined as first-line levers that recalibrate the HPA axis, autonomic system, and inflammatory pathways that influence menopausal symptom expression.

Attendees will learn how to evaluate hormone metabolites to better understand tissue-level signaling rather than relying solely on serum parent hormones. The presentation connects these biochemical insights with clinical patterns such as estrogen dominance, low progesterone resilience, cortisol-driven vasomotor symptoms, mitochondrial-driven fatigue, and inflammatory or histamine-related reactivity.

Ultimately, this session offers a comprehensive, actionable framework for practitioners seeking to support women through menopause with precision, depth, and personalization. By sequencing interventions thoughtfully and addressing underlying physiological imbalances first, clinicians can enhance therapeutic outcomes, minimize symptom burden, and help women move through midlife with strength, clarity, and vitality.